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Finally, a number of inherited mutations in genes involved in carnitine shuttling and fatty acid oxidation pathways do not systematically result in carnitine depletion (such that carnitine supplementation may not help mitigate the symptoms) but lead to abnormal profiles of acylcarnitine esters in blood (35, 40)

In addition, overexpression of parkin in APP/PS1 mouse models restores impaired long-term potentiation (LTP) and rescues behavioral abnormalities by reducing A load and neuroinflammation (Hong et al., 2014)

Inflammopharmacology 26, 10931101

[DOI] [PubMed] [Google Scholar] 118.Russell J., Maguire S., Hunt G.E., Kesby A., Suraev A., Stuart J., Booth J., McGregor I.S

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